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  3. MOCK2CC Hypotyhroidism

MOCK2CC Hypotyhroidism

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  • A Online
    A Online
    admin
    wrote last edited by admin
    #1

    Stem
    A 65-year-old lady, presented with long standing goitre, malaise & chronic fatigue. FBC & full biochemical profile reviewed as part of her assessment & MAC showed low T3 & T4, high TSH along with macrocytic anaemia.

    What's the most likely clinical diagnosis?

    Hypothyroidism

    Describe the physiological regulation of thyroid hormones

    The physiological regulation of thyroid hormones involves a complex feedback loop involving the hypothalamus, pituitary gland, and thyroid gland:
    Hypothalamus – TRH: Releases thyrotropin‑releasing hormone (TRH), stimulating the pituitary.
    Pituitary – TSH: TRH triggers release of thyroid‑stimulating hormone (TSH), which stimulates the thyroid gland.
    Thyroid – T3 & T4: TSH stimulates production and release of thyroxine (T4) and triiodothyronine (T3).
    Negative feedback: High levels of T3/T4 inhibit TRH and TSH release, maintaining balance.

    What are differences between T3 & T4

    T3 is more biologically active than T4.
    T4 has greater protein‑binding capacity than T3.

    Talk about Synthesis of T3 & T4

    Iodide ions enter thyroid follicular cells via active pumping.
    Iodide is converted to iodine by TPO (thyroid peroxidase).
    Iodine combines with tyrosine to form MIT (monoiodotyrosine) and DIT (diiodotyrosine).
    MIT + DIT = T3
    DIT + DIT = T4

    Signs of hypothyroidism

    Weight gain
    Memory loss
    Cold intolerance
    Constipation
    Myxoedema
    Bradycardia
    Muscle weakness
    Pretibial oedema
    Dry skin

    Causes of hypothyroidism
    Primary hypothyroidism

    Iatrogenic
    -Surgery
    -Radioiodine therapy
    -Drugs
    Autoimmune (Hashimoto’s)
    Iodine deficiency
    Idiopathic
    Genetic defects in thyroid development
    Thyroid hormone resistance syndrome (THRB) (rare)
    Congenital biosynthetic defect (dyshormonogenetic goitre) (rare)
    Transient thyroiditis (De Quervain’s thyroiditis)
    Infiltrative (amyloidosis, sarcoidosis)

    Secondary hypothyroidism

    Pituitary failure (rare)
    Hypothalamic failure (rare)

    If hypothyroidism is due to hypopituitarism, how could TFT differ?

    This will be secondary hypothyroidism:
    Low TSH
    Low T3 and T4

    From the FBC findings given, explain the cause

    Patient has macrocytic anaemia due to antibodies directed against parietal cells, which are responsible for secretion of intrinsic factor that helps in absorption of vitamin B12.
    I.e. autoimmune hypothyroidism associated with pernicious anaemia.

    She was put on medical therapy; she is not compliant. Which teams should be involved?

    Endocrinologist
    Her GP
    Family members

    Problems associated with emergency surgery in hypothyroid patients

    Pre‑operative
    Anaemia
    Increased risk of ischaemic heart disease
    Increased sensitivity to medications, anaesthetic drugs, and narcotics
    Intra‑operative
    Airway compromise
    Coagulopathy
    Post‑operative
    Myxoedema coma
    Delayed recovery
    Poor wound healing

    How to improve compliance with thyroid replacement therapy

    Discuss with the patient the reason for non‑compliance
    Communication with GP and family members
    Medical follow‑up
    Simple regimen: single dose in the early morning

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