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Gastric outlet obstruction

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  • A Offline
    A Offline
    admin
    wrote last edited by admin
    #1
    1. What are the common causes of gastric outlet obstruction in adults?

    The causes can be divided into benign and malignant.
    Benign
    Peptic ulcer disease causing pyloric stenosis
    Chronic pancreatitis
    Caustic ingestion
    Post-surgical strictures
    Crohn’s disease
    Malignant
    Distal gastric cancer (most common modern cause)
    Pancreatic head cancer
    Duodenal carcinoma
    Periampullary tumours
    Historically peptic ulcer disease was the commonest cause, but malignancy is now more common.

    1. Why does gastric outlet obstruction cause vomiting of undigested food?

    The obstruction occurs at the pylorus or proximal duodenum, preventing gastric emptying.
    Food therefore remains in the stomach for prolonged periods and is vomited before reaching the small intestine, so it appears undigested.

    1. What electrolyte and acid–base abnormalities are classically seen?

    The classical abnormality is:
    Hypochloraemic
    Hypokalaemic
    Metabolic alkalosis
    Often associated with hyponatraemia and volume depletion.

    1. Why does vomiting lead to metabolic alkalosis?

    Gastric secretions contain hydrochloric acid (HCl).
    Vomiting causes loss of:
    Hydrogen ions
    Chloride ions
    Loss of hydrogen ions leads to metabolic alkalosis, and loss of chloride contributes to hypochloraemia, which also impairs renal bicarbonate excretion and worsens the alkalosis.

    1. Why does hypokalaemia occur in gastric outlet obstruction?

    Hypokalaemia occurs due to:
    Renal potassium loss from activation of the renin–angiotensin–aldosterone system due to dehydration.
    Hydrogen–potassium exchange in the kidney during metabolic alkalosis, where potassium is excreted to retain hydrogen ions.

    1. What clinical signs might you find on examination?

    Possible signs include:
    Epigastric distension
    Succussion splash (if stomach contains retained fluid)
    Visible gastric peristalsis
    Signs of dehydration (tachycardia, hypotension)
    Possible weight loss if chronic

    1. What investigations would you perform to confirm the diagnosis?

    Investigations include:
    Blood tests: FBC, U&E, LFTs, ABG
    Nasogastric aspiration (large residual volume)
    Upper GI endoscopy (OGD) – confirms obstruction and allows biopsy
    CT abdomen – helps identify malignancy or extrinsic compression
    OGD is the key diagnostic test.

    1. How would you manage this patient initially?

    Initial management is resuscitation and decompression:
    Nil by mouth
    Nasogastric tube for gastric decompression
    IV fluids to correct dehydration
    Correct electrolyte abnormalities
    IV proton pump inhibitor
    Arrange urgent endoscopy
    Definitive treatment depends on the cause (e.g., endoscopic dilation, surgery, or oncological treatment).

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