<?xml version="1.0" encoding="UTF-8"?><rss xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:content="http://purl.org/rss/1.0/modules/content/" xmlns:atom="http://www.w3.org/2005/Atom" version="2.0"><channel><title><![CDATA[MockCC37 Sigmoid volvulus]]></title><description><![CDATA[<p dir="auto">Stem : 85-year-old lady living in care home. She uses a wheelchair and has long-standing constipation. She presented in the A&amp;E with abdominal pain, constipation, marked abdominal distention, vomiting and dehydration<br />
Labs: Na 120, ABG: PH 7.5, PCO₂ 4.2.</p>
<p dir="auto"><img src="/assets/uploads/files/1785574679012-3a739803-b71f-43e0-94e5-70404dda3e34-image.jpeg" alt="3a739803-b71f-43e0-94e5-70404dda3e34-image.jpeg" class=" img-fluid img-markdown" /></p>
<p dir="auto">What are the possible causes of this clinical picture?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Given the age of the patient, past medical history and long history of constipation, as well as AXR showing coffee-bean shape so I should suspect sigmoid volvulus.</p></blockquote>
<p dir="auto">What is your interpretation of this ABG?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Metabolic alkalosis</p></blockquote>
<p dir="auto">Explain the biochemical abnormalities, why alkalosis?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Loss of H⁺ in the vomitus.<br />
Increase bicarbonate uptake in renal tubules in response to loss of chloride in order to maintain electrochemical neutrality.<br />
Retention of bicarbonate-rich pancreatic juice due to loss of acid load in the duodenum.</p></blockquote>
<p dir="auto">Why hyponatremia?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Kidneys excrete more NaHCO₃ to reduce blood alkalinity.<br />
Na loss in vomitus also.</p></blockquote>
<p dir="auto">Why hypochloraemia?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Due to loss of chloride (HCL) in the vomitus.</p></blockquote>
<p dir="auto">Why hypokalaemia?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Due to increased aldosterone in response to hypovolemia which will absorb Na and water in exchange of K in the kidney.<br />
Loss of K⁺ in vomitus.</p></blockquote>
<p dir="auto">Why raised Bicarbonate?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Due to increased bicarbonate uptake in renal tubules in response to loss of chloride in order to maintain electrochemical neutrality.</p></blockquote>
<p dir="auto">Why aciduria?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Loss of H⁺ in urine in exchange of Na⁺</p></blockquote>
<p dir="auto">Is NaHCO₃ excreted as a whole ion?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Yes</p></blockquote>
<p dir="auto">How would manage this patient?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">I would manage the patient according to CCriSP protocol (ABC)<br />
Keep NBM, NG tube<br />
Catheter and fluid balance chart<br />
IV access<br />
Start IV fluid (normal saline + kcl)<br />
Monitor vital signs (pulse, BP, UOP and capillary refill time and mental state)<br />
Early detorsion with rigid sigmoidoscopy +/- insertion of flatus tube (rectal tube)<br />
I will first try rigid sigmoidoscopy, if fails then I will do flexible sigmoidoscopy<br />
Followed by elective surgery to correct underlying cause</p></blockquote>
<p dir="auto">What are the symptoms and signs of sigmoid volvulus?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Constipation<br />
Abdominal distension<br />
Abdominal pain<br />
Late vomiting (Feculent vomiting)<br />
Signs of dehydration (tachycardia, hypotension, oliguria)<br />
Distended tender abdomen</p></blockquote>
<p dir="auto">What are its complications?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Bowel ischemia and gangrene<br />
Perforation<br />
Peritonitis and sepsis</p></blockquote>
<p dir="auto">Who are you going involve in the care of this lady?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Colorectal Surgeon<br />
Anaesthetist<br />
ITU<br />
Cardiologist</p></blockquote>
<p dir="auto">Define volvulus?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">A volvulus is a twisting or axial rotation of a portion of bowel around its mesentery, can lead to obstruction of the lumen and vascular occlusion.<br />
The rotation causes obstruction of the lumens (&gt;180° torsion) and if tight enough also causes vascular occlusion in the mesentery (&gt;360° torsion)</p></blockquote>
<p dir="auto">Risk factors of volvulus</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Increasing age<br />
Male gender<br />
Chronic constipation or laxative use<br />
Previous abdominal operations<br />
Neuropsychiatric disorders<br />
Nursing home resident</p></blockquote>
<p dir="auto">What are the different surgical options for treating a patient with sigmoid volvulus?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Hartmann’s procedure with end colostomy or Mikulicz procedure.<br />
Laparotomy with manual detorsion and assess for viability. If viable do sigmoidopexy to the lateral wall of abdomen or pelvis.</p></blockquote>
]]></description><link>https://isurg.org/topic/512/mockcc37-sigmoid-volvulus</link><generator>RSS for Node</generator><lastBuildDate>Sat, 01 Aug 2026 23:48:11 GMT</lastBuildDate><atom:link href="https://isurg.org/topic/512.rss" rel="self" type="application/rss+xml"/><pubDate>Sat, 01 Aug 2026 09:02:12 GMT</pubDate><ttl>60</ttl><item><title><![CDATA[Reply to MockCC37 Sigmoid volvulus on Sat, 01 Aug 2026 09:05:49 GMT]]></title><description><![CDATA[<p dir="auto">What are the clinical manifestations of hyponatremia?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">Arrhythmia and cardiac arrest<br />
Confusion<br />
Agitation<br />
Fits<br />
Reduced GCS</p></blockquote>
<p dir="auto">What are the causes of hyponatremia?</p>
<blockquote class="spoiler border border-warning"><button class="btn btn-sm btn-ghost border">Spoiler</button><p class="d-none mt-3 text-sm">According to serum osmolarity, hyponatremia can be divided into: Isotonic, Hypotonic, Hypertonic<br />
Hypotonic hyponatremia can be divided according to volume status:<br />
Hypovolemic hyponatremia e.g. diarrhoea, vomiting and diuretics<br />
Euvolemic hyponatremia e.g. SIADH, hypothyroidism<br />
Hypervolemic hyponatremia e.g. heart failure<br />
Pseudohyponatremia: Multiple myeloma</p></blockquote>
]]></description><link>https://isurg.org/post/777</link><guid isPermaLink="true">https://isurg.org/post/777</guid><dc:creator><![CDATA[admin]]></dc:creator><pubDate>Sat, 01 Aug 2026 09:05:49 GMT</pubDate></item></channel></rss>